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Introduction Unrepaired DNA lesions and deficit in pathways repairing DNA have been documented in several neurodegenerative diseases, including Alzheimers disease (AD
Kuah E, Toh S, Yee J et al (2016) Enzyme mimics: advances and applications
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Low Wang said doctors have learned a great deal in recent years about genetic causes of obesity
Because IGF-1 is a central mediator of GHs effects, even minor deviations can disproportionately alter metabolism, growth, and repair processes
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